Skip to main content
OREGON STATE UNIVERSITY
Open search box
Linus Pauling Institute
Toggle menu
Go to search page
Search Field
Exit Search
About the LPI
Faculty and Staff
Research
Outreach
Micronutrients for Health
Diet and Optimum Health Conference
Donate
About the LPI
Faculty and Staff
Research
Outreach
Diet and Optimum Health Conference
Donate
Biblio
Found 48 results
Author
[
Keyword
]
Title
Type
Year
Filters:
Author
is
Dixon, Brian M
[Clear All Filters]
A
B
C
D
E
F
G
H
I
J
K
L
M
N
O
P
Q
R
S
T
U
V
W
X
Y
Z
Iodoacetic Acid
Dixon BM
,
Heath S-HD
,
Kim R
,
Suh JH
,
Hagen TM
. 2008.
Assessment of endoplasmic reticulum glutathione redox status is confounded by extensive ex vivo oxidation.
.
Antioxid Redox Signal. 10(5):963-72.
Introns
Li Q
,
Dixon BM
,
Al-Fageeh M
,
Blum CA
,
Dashwood RH
. 2002.
Sequencing of the rat beta-catenin gene (Ctnnb1) and mutational analysis of liver tumors induced by 2-amino-3-methylimidazo[4,5-f]quinoline.
.
Gene. 283(1-2):255-62.
In Vitro Techniques
Suh JH
,
Shenvi SV
,
Dixon BM
,
Liu H
,
Jaiswal AK
,
Liu R-M
,
Hagen TM
. 2004.
Decline in transcriptional activity of Nrf2 causes age-related loss of glutathione synthesis, which is reversible with lipoic acid.
.
Proc Natl Acad Sci U S A. 101(10):3381-6.
Glutathione Disulfide
Dixon BM
,
Heath S-HD
,
Kim R
,
Suh JH
,
Hagen TM
. 2008.
Assessment of endoplasmic reticulum glutathione redox status is confounded by extensive ex vivo oxidation.
.
Antioxid Redox Signal. 10(5):963-72.
Glutathione
Suh JH
,
Shenvi SV
,
Dixon BM
,
Liu H
,
Jaiswal AK
,
Liu R-M
,
Hagen TM
. 2004.
Decline in transcriptional activity of Nrf2 causes age-related loss of glutathione synthesis, which is reversible with lipoic acid.
.
Proc Natl Acad Sci U S A. 101(10):3381-6.
Dixon BM
,
Heath S-HD
,
Kim R
,
Suh JH
,
Hagen TM
. 2008.
Assessment of endoplasmic reticulum glutathione redox status is confounded by extensive ex vivo oxidation.
.
Antioxid Redox Signal. 10(5):963-72.
Glutamate-Cysteine Ligase
Suh JH
,
Shenvi SV
,
Dixon BM
,
Liu H
,
Jaiswal AK
,
Liu R-M
,
Hagen TM
. 2004.
Decline in transcriptional activity of Nrf2 causes age-related loss of glutathione synthesis, which is reversible with lipoic acid.
.
Proc Natl Acad Sci U S A. 101(10):3381-6.
Genes
Li Q
,
Dixon BM
,
Al-Fageeh M
,
Blum CA
,
Dashwood RH
. 2002.
Sequencing of the rat beta-catenin gene (Ctnnb1) and mutational analysis of liver tumors induced by 2-amino-3-methylimidazo[4,5-f]quinoline.
.
Gene. 283(1-2):255-62.
Exons
Li Q
,
Dixon BM
,
Al-Fageeh M
,
Blum CA
,
Dashwood RH
. 2002.
Sequencing of the rat beta-catenin gene (Ctnnb1) and mutational analysis of liver tumors induced by 2-amino-3-methylimidazo[4,5-f]quinoline.
.
Gene. 283(1-2):255-62.
Enzyme Inhibitors
Dixon BM
,
Heath S-HD
,
Kim R
,
Suh JH
,
Hagen TM
. 2008.
Assessment of endoplasmic reticulum glutathione redox status is confounded by extensive ex vivo oxidation.
.
Antioxid Redox Signal. 10(5):963-72.
Endoplasmic Reticulum
Dixon BM
,
Heath S-HD
,
Kim R
,
Suh JH
,
Hagen TM
. 2008.
Assessment of endoplasmic reticulum glutathione redox status is confounded by extensive ex vivo oxidation.
.
Antioxid Redox Signal. 10(5):963-72.
DNA-Binding Proteins
Suh JH
,
Shenvi SV
,
Dixon BM
,
Liu H
,
Jaiswal AK
,
Liu R-M
,
Hagen TM
. 2004.
Decline in transcriptional activity of Nrf2 causes age-related loss of glutathione synthesis, which is reversible with lipoic acid.
.
Proc Natl Acad Sci U S A. 101(10):3381-6.
DNA, Complementary
Li Q
,
Dixon BM
,
Al-Fageeh M
,
Blum CA
,
Dashwood RH
. 2002.
Sequencing of the rat beta-catenin gene (Ctnnb1) and mutational analysis of liver tumors induced by 2-amino-3-methylimidazo[4,5-f]quinoline.
.
Gene. 283(1-2):255-62.
DNA
Li Q
,
Dixon BM
,
Al-Fageeh M
,
Blum CA
,
Dashwood RH
. 2002.
Sequencing of the rat beta-catenin gene (Ctnnb1) and mutational analysis of liver tumors induced by 2-amino-3-methylimidazo[4,5-f]quinoline.
.
Gene. 283(1-2):255-62.
Cytoskeletal Proteins
Li Q
,
Dixon BM
,
Al-Fageeh M
,
Blum CA
,
Dashwood RH
. 2002.
Sequencing of the rat beta-catenin gene (Ctnnb1) and mutational analysis of liver tumors induced by 2-amino-3-methylimidazo[4,5-f]quinoline.
.
Gene. 283(1-2):255-62.
beta Catenin
Li Q
,
Dixon BM
,
Al-Fageeh M
,
Blum CA
,
Dashwood RH
. 2002.
Sequencing of the rat beta-catenin gene (Ctnnb1) and mutational analysis of liver tumors induced by 2-amino-3-methylimidazo[4,5-f]quinoline.
.
Gene. 283(1-2):255-62.
Base Sequence
Li Q
,
Dixon BM
,
Al-Fageeh M
,
Blum CA
,
Dashwood RH
. 2002.
Sequencing of the rat beta-catenin gene (Ctnnb1) and mutational analysis of liver tumors induced by 2-amino-3-methylimidazo[4,5-f]quinoline.
.
Gene. 283(1-2):255-62.
Antioxidants
Suh JH
,
Shenvi SV
,
Dixon BM
,
Liu H
,
Jaiswal AK
,
Liu R-M
,
Hagen TM
. 2004.
Decline in transcriptional activity of Nrf2 causes age-related loss of glutathione synthesis, which is reversible with lipoic acid.
.
Proc Natl Acad Sci U S A. 101(10):3381-6.
Animals
Suh JH
,
Shenvi SV
,
Dixon BM
,
Liu H
,
Jaiswal AK
,
Liu R-M
,
Hagen TM
. 2004.
Decline in transcriptional activity of Nrf2 causes age-related loss of glutathione synthesis, which is reversible with lipoic acid.
.
Proc Natl Acad Sci U S A. 101(10):3381-6.
Dixon BM
,
Heath S-HD
,
Kim R
,
Suh JH
,
Hagen TM
. 2008.
Assessment of endoplasmic reticulum glutathione redox status is confounded by extensive ex vivo oxidation.
.
Antioxid Redox Signal. 10(5):963-72.
Li Q
,
Dixon BM
,
Al-Fageeh M
,
Blum CA
,
Dashwood RH
. 2002.
Sequencing of the rat beta-catenin gene (Ctnnb1) and mutational analysis of liver tumors induced by 2-amino-3-methylimidazo[4,5-f]quinoline.
.
Gene. 283(1-2):255-62.
Amino Acid Sequence
Li Q
,
Dixon BM
,
Al-Fageeh M
,
Blum CA
,
Dashwood RH
. 2002.
Sequencing of the rat beta-catenin gene (Ctnnb1) and mutational analysis of liver tumors induced by 2-amino-3-methylimidazo[4,5-f]quinoline.
.
Gene. 283(1-2):255-62.
Aging
Suh JH
,
Shenvi SV
,
Dixon BM
,
Liu H
,
Jaiswal AK
,
Liu R-M
,
Hagen TM
. 2004.
Decline in transcriptional activity of Nrf2 causes age-related loss of glutathione synthesis, which is reversible with lipoic acid.
.
Proc Natl Acad Sci U S A. 101(10):3381-6.
Pages
« first
‹ previous
1
2
Donate Today!
Get Updates from the Institute